UN CASO DI FLUTTER ATRIALE

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1 UN CASO DI FLUTTER ATRIALE
MINIMASTER DI ELETTROCARDIOGRAFIA L’ECG NELLE TACHICARDIE SOPRAVENTRICOLARI UN CASO DI FLUTTER ATRIALE DISCUSSANT : G. LEONE

2 Caso clinico G.T. uomo di anni 55
Affetto da pervietà interatriale tipo ostium secundum corretta chirurgicamente nel 1987 Iperteso in trattamento con ibesartan Ecocardio: ingrandimento biatriale modesto. Setto Interatriale senza soluzione di continuità. Setto IV lievemente ipertrofico e discinetico. Normali le cavità ventricolari sx. Nel 1996 inizia a manifestare episodi ricorrenti di cardiopalmo per flutter atriale. Iniziato trattamento con antiaritmici : flecainide,propafenone, amiodarone. Nel 1997 ricovero per ablazione.

3 Ecg di base

4 Durante la procedura di ablazione si osserva :
Ciclo F-F di 250 msec Onde F negative in D2,D3,AVF, positive in V1

5 Come classificare questo flutter?
Dall’esame del ciclo e della morfologia dell’onda F osserviamo: Ciclo F-F di 250 msec Onde F negative in D2,D3,AVF, positive in V1

6 TYPICAL ATRIAL FLUTTER
F Arribas, F. Cosìo ATRIAL FLUTTER ABLATION: role of endocardial mapping in J. Farrè-C. Moro: Ten years of RA, Futura 1998

7 Questo pattern a che cosa è ascrivibile?
Flutter atriale tipico antiorario.

8 Typical atrial flutter
Typical atrial flutter: a macro-re-entry atrial regular tachycardia, either present at baseline or inducible by atrial stimulation, where the involvement of the inferior vena cava — tricuspid annulus isthmus could be proven by concealed entrainment. The arrhythmia was defined as counterclockwise or clockwise in accordance with the direction of the activation wavefront recorded on the tricuspid annulus catheter.

9 Durante la procedura di ablazione si osserva :
Ciclo F-F di 250 msec Onde F negative in D2,D3,AVF, positive in V1

10 Subito dopo, durante la procedura, si osserva :
Ciclo F-F 250 msec Onda F positiva in D1,D2,difasica in aVL, Isoelettrica in aVF, negativa in V1.

11 Il confronto ci fa apprezzare meglio le differenze:

12 Di cosa si tratta? Possiamo fare delle ipotesi:

13 Di cosa si tratta? Reverse typical atrial flutter
Double loop intra-atrial Lower loop flutter Lesion macroreentrant tachycardia (Right atrial free wall macroreentry without atriotomy) Left atrial macroreentrant tachycardia

14 REVERSE TYPICAL ATRIAL FLUTTER
F Arribas, F. Cosìo ATRIAL FLUTTER ABLATION: role of endocardial mapping in J. Farrè-C. Moro: Ten years of RA, Futura 1998

15 Clinical and Echocardiographic Characteristics of Two Types of CWID

16 Morphologic variations of clockwise isthmus-dependent atrial flutter on 12-lead surface electrocardiogram: (A) Type 1: F+(n) or F+ in II, III, aVF, and V6; F- in V1, narrow F-wave/distinct isoelectric segment

17 (B) Type 2: f-/F+9(n) or F+(n) inferiorly and V6, isoelectric in V1, with a broad F-wave and no distinct isoelectric segment.

18 LOWER LOOP FLUTTER Zhang et al Clockwise Lower Loop Reentry
Circulation 2004;109; ;

19 Left atrial macroreentrant tachycardia
(Circulation 2004;109: )

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21 Il passaggio da una deflessione negativa nelle derivazioni inferiori a deflessioni positive indica che il cambio di polarità all’ecg è associato con una alterata attivazione settale che cambia da caudocraniale a craniocaudale e riflette probabilmente una simile modificazione nella attivazione dell’atrio sinistro.

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23 This article describes the characteristics of dual-loop, figure-8 atrial reentry. All occurred in patients previously operated on for surgical closure of an ostium secundum atrial septal defect and required ablation of 2 isthmuses to be curative. The simultaneous coexistence of both loops was proved by comprehensive 3D mapping and transformation to a single loop circuit with appropriately targeted RF application. The double-loop mechanism was revealed on ECG only by transection of 1 loop. The ECG transformation was instantaneous and without an intervening pause, which might suggest termination followed by induction. The mapping of transformation of a superior-axis negativedeflection flutter (in inferior leads) to a positive-deflection flutter (inferior P-wave axis) with a periatriotomy reentry circuit indicates that the polarity change on the surface ECG is associated with altered septal activation from caudocranial to craniocaudal and possibly a similar change in left atrial activation. In all the above cases, ablation was begun in the cavotricuspid isthmus; however, if we had begun by ablating the IVC-atriotomy isthmus, in all likelihood no significant surface ECG changes would have occurred because the resulting change in atrial activation would be limited to only a part of the lateral and posterior RA, which appears to be silent on the ECG.

24 Transection of 1 loop (peritricuspid) allowed unopposed atrial activation by the other loop (periatriotomy) to be evident on the surface ECG. This novel mechanism of transformation of a reentrant tachycardia ECG may also be considered the gold standard test for a true figure-8 reentry circuit in which the remaining loop is capable of independent stability as opposed to a figure-8 activation pattern. The double-loop mechanism was revealed on ECG only by transection of 1 loop. The ECG transformation was instantaneous and without an intervening pause, which might suggest termination followed by induction. The mapping of transformation of a superior-axis negativedeflection flutter (in inferior leads) to a positive-deflection flutter (inferior P-wave axis) with a periatriotomy reentry circuit indicates that the polarity change on the surface ECG is associated with altered septal activation from caudocranial to craniocaudal and possibly a similar change in left atrial activation. In all the above cases, ablation was begun in the cavotricuspid isthmus; however, if we had begun by ablating the IVC-atriotomy isthmus, in all likelihood no significant surface ECG changes would have occurred because the resulting change in atrial activation would be limited to only a part of the lateral and posterior RA, which appears to be silent on the ECG.

25 Simultaneous periatriotomy loop activation is not
clearly evident on the surface ECG and that similarly posterior RA activation in typical flutter may not contribute to the surface ECG tracing. The double-loop mechanism was revealed on ECG only by transection of 1 loop. The ECG transformation was instantaneous and without an intervening pause, which might suggest termination followed by induction. The double-loop mechanism was revealed on ECG only by transection of 1 loop. The ECG transformation was instantaneous and without an intervening pause, which might suggest termination followed by induction. The mapping of transformation of a superior-axis negativedeflection flutter (in inferior leads) to a positive-deflection flutter (inferior P-wave axis) with a periatriotomy reentry circuit indicates that the polarity change on the surface ECG is associated with altered septal activation from caudocranial to craniocaudal and possibly a similar change in left atrial activation. In all the above cases, ablation was begun in the cavotricuspid isthmus; however, if we had begun by ablating the IVC-atriotomy isthmus, in all likelihood no significant surface ECG changes would have occurred because the resulting change in atrial activation would be limited to only a part of the lateral and posterior RA, which appears to be silent on the ECG.

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30 This is the first reported case of dual-loop intra-atrial re-entry tachycardia in a patient
with ischaemic cardiomyopathy. In our case, two tachycardia re-entry loops were connected (‘figure-of-eight’) such that one tachycardia could be transformed into the other tachycardia without pause or atrial extra-systole. This re-entry loop may possibly be related to scar tissue due to multiple prior coronary bypass procedures.

31 Shah et al.[5] described dual-loop intra-atrial re-entry
tachycardia in a series of five patients. However, in contrast to our case, all of their patients had prior atrial septal defect repair. Of interest all their patients had counter-clockwise TA/IVC isthmus dependent atrial flutter and a second loop related to an atriotomy/scar tissue in the right atrial free wall. Similar to our case, ablation at two distinct sites was needed to cure these two re-entry tachycardias.

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